- Title: Subtitle
- Abstract 4695: Dual targeting of MEK and PI3K pathways can act via tumor-intrinsic mechanisms to overcome resistance and tumor-extrinsic mechanisms to modulate immunity and limit cancer cachexia
- Creators
- Jennifer Yang - The Ohio State UniversityErin Talbert - The Ohio State UniversityOmar Elnaggar - The Ohio State UniversityPriyani Rajasekera - The Ohio State UniversityThomas Mace - The Ohio State UniversityMatthew Farren - The Ohio State UniversityZheng Che - The Ohio State UniversityBenjamin Swanson - The Ohio State UniversityGregory Young - The Ohio State UniversityEricka Haverick - The Ohio State UniversityCynthia Timmers - The Ohio State UniversityMark Bloomston - The Ohio State UniversityTanios Bekaii-Saab - The Ohio State UniversityDenis Guttridge - The Ohio State UniversityGregory Lesinski - The Ohio State University
- Resource Type
- Abstract
- Publication Details
- Cancer research (Chicago, Ill.), Vol.75(15 Supplement), pp.4695-4695
- DOI
- 10.1158/1538-7445.AM2015-4695
- ISSN
- 0008-5472
- eISSN
- 1538-7445
- Language
- English
- Date published
- 08/01/2015
- Academic Unit
- Internal Medicine; Fraternal Order of Eagles Diabetes Research Center; Health and Human Physiology
- Record Identifier
- 9984267244702771
Abstract
Abstract 4695: Dual targeting of MEK and PI3K pathways can act via tumor-intrinsic mechanisms to overcome resistance and tumor-extrinsic mechanisms to modulate immunity and limit cancer cachexia
Cancer research (Chicago, Ill.), Vol.75(15 Supplement), pp.4695-4695
08/01/2015
DOI: 10.1158/1538-7445.AM2015-4695
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