- Title: Subtitle
- CaMKII oxidation causes increased atrial fibrillation in diabetic mice
- Creators
- Olurotimi Mesubi - Johns Hopkins University School of MedicineAdam Rokita - Division of Cardiovascular Medicine and Cardiovascular Research Center, Carver College of Medicine, Iowa City, IA, USANeha Abrol - Johns Hopkins University School of MedicineYuejin Wu - Johns Hopkins University School of MedicineBiyi Chen - Division of Cardiovascular Medicine and Cardiovascular Research Center, Carver College of Medicine, Iowa City, IA, USAQinchuan Wang - Johns Hopkins University School of MedicineJonathan Granger - Johns Hopkins University School of MedicineElizabeth Luczak - Johns Hopkins University School of MedicineLars Maier - Division of Cardiology and Pneumology, German Heart Center, University Hospital Goettingen, Goettingen, Germany, USAXander Wehrens - Baylor College of MedicineJoel Pomenrantz - Johns Hopkins University School of MedicineLong-Sheng Song - Division of Cardiovascular Medicine and Cardiovascular Research Center, Carver College of Medicine, Iowa City, IA, USAGerald Hart - Johns Hopkins University School of MedicineMark Anderson - Johns Hopkins University School of Medicine
- Resource Type
- Abstract
- Publication Details
- Journal of molecular and cellular cardiology, Vol.112, pp.161-162
- Publisher
- Elsevier Ltd
- DOI
- 10.1016/j.yjmcc.2017.07.092
- ISSN
- 0022-2828
- eISSN
- 1095-8584
- Language
- English
- Date published
- 11/2017
- Academic Unit
- Cardiovascular Medicine; Fraternal Order of Eagles Diabetes Research Center; Internal Medicine; Biochemistry and Molecular Biology
- Record Identifier
- 9984297352902771
Abstract
CaMKII oxidation causes increased atrial fibrillation in diabetic mice
Journal of molecular and cellular cardiology, Vol.112, pp.161-162
11/2017
DOI: 10.1016/j.yjmcc.2017.07.092
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