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A Case of Bow Hunter's Syndrome Caused by C3/4 Facet Joint Hypertrophy: Hemodynamic Complexity and Surgical Strategy
Journal article   Open access

A Case of Bow Hunter's Syndrome Caused by C3/4 Facet Joint Hypertrophy: Hemodynamic Complexity and Surgical Strategy

Satoka Shidoh, Katherine J. Jensen, Clayton L. Rosinski, Kathleen E. Dlouhy and Satoshi Yamaguchi
NMC case report journal, Vol.13, pp.457-462
09/17/2026
DOI: 10.2176/jns-nmc.2026-0089
PMCID: PMC13668433
PMID: 42877945
url
https://doi.org/10.2176/jns-nmc.2026-0089View
Published (Version of record) Open Access

Abstract

Bow Hunter's syndrome results from rotational insufficiency of the vertebral artery. Though traditionally associated with C1-C2 level compression, subaxial cervical causes are increasingly recognized. We report a 68-year-old man with codominant vertebral arteries presenting with dizziness and transient visual disturbances during head rotation. Imaging showed severe focal stenosis caused by an osteophyte at the left C3-C4 facet joint. Dynamic angiography revealed that the left vertebral artery was nearly occluded during rotation; despite faint distal residual flow, contrast failed to reach the basilar artery. The patient underwent a left C3-C4 facetectomy and posterior C2-C5 fusion. Intraoperative angiography confirmed restoration of antegrade flow, and the patient's symptoms resolved postoperatively. Identifying the underlying hemodynamic mechanism and selecting an appropriate surgical route are important for the successful management of this condition.

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