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A Genotype-Phenotype Analysis of Glutathione Peroxidase 4 in Human Atrial Myocardium and Its Association with Postoperative Atrial Fibrillation
Journal article   Open access   Peer reviewed

A Genotype-Phenotype Analysis of Glutathione Peroxidase 4 in Human Atrial Myocardium and Its Association with Postoperative Atrial Fibrillation

Islam A Berdaweel, Alexander A Hart, Andrew J Jatis, Nathan Karlan, Shahab A Akhter, Marie E Gaine, Ryan M Smith and Ethan J Anderson
Antioxidants, Vol.11(4), p.721
04/06/2022
DOI: 10.3390/antiox11040721
PMCID: PMC9026099
PMID: 35453406
url
https://doi.org/10.3390/antiox11040721View
Published (Version of record) Open Access

Abstract

Heterogeneity in the incidence of postoperative atrial fibrillation (POAF) following heart surgery implies that underlying genetic and/or physiological factors impart a higher risk of this complication to certain patients. Glutathione peroxidase-4 (GPx4) is a vital selenoenzyme responsible for neutralizing lipid peroxides, mediators of oxidative stress known to contribute to postoperative arrhythmogenesis. Here, we sought to determine whether single nucleotide variants are associated with POAF, and whether any of these variants are linked with altered enzyme content or activity in myocardial tissue. Sequencing analysis was performed across the coding region within chromosome 19 from a cohort of patients (N = 189) undergoing elective coronary artery bypass graft (-/+ valve) surgery. GPx4 enzyme content and activity were also analyzed in matching samples of atrial myocardium from these patients. Incidence of POAF was 25% in this cohort. Five variants were associated with POAF risk (permutated ≤ 0.05), and eight variants associated with altered myocardial GPx4 content and activity ( < 0.05). One of these variants (rs713041) is a well-known modifier of cardiovascular disease risk. Collectively, these findings suggest variants are potential risk modifiers and/or predictors of POAF. Moreover, they illustrate a genotype-phenotype link with this selenoenzyme, which will inform future mechanistic studies.
Biomarkers postoperative atrial fibrillation glutathione peroxidase-4 single nucleotide variants reactive oxygen species

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