ARHGAP29 promotes keratinocyte proliferation and migration in vitro and is dispensable for in vivo wound healing

The authors would like to thank Dr. Paul Martin for sharing ARHGAP29 ideas and unpublished data, Drs. Eric Taylor, Adam Rauckhorst and Botond Bonfi for critical discussion related to CRISPR, and all Dunnwald lab and Tootle lab members for continuous input. The technical expertise of Annemarie Carver, Campbell Mitvalsky and members of the W.M. Keck Dynamic Image Analysis facility in the Biology department of the University of Iowa is greatly appreciated. The data presented herein were obtained using the Flow Cytometry Facility, University of Iowa Central Microscopy Research Facility, University of Iowa Viral Vector Core, and the Genomics Division of the Iowa Institute of Human Genetics which are all supported by the Carver College of Medicine at the University of Iowa. This work was supported by funding from the National Institute of Health (NIH, R01-AR067739, M. D.; T32-GM145441, E. A.), the American Association for Anatomy Fellows Grant Program (M. D.), the Department of Anatomy and Cell Biology, the Carver College of Medicine (M. D., A. S., and A. K.) and the Office of Undergraduate Research (B. G.) from the University of Iowa.