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ASICs Mediate Pain and Inflammation in Musculoskeletal Diseases
Journal article   Open access   Peer reviewed

ASICs Mediate Pain and Inflammation in Musculoskeletal Diseases

Ramy E Abdelhamid and Kathleen A Sluka
Physiology (Bethesda, Md.), Vol.30(6), pp.449-459
11/2015
DOI: 10.1152/physiol.00030.2015
PMCID: PMC4747907
PMID: 26525344
url
https://doi.org/10.1152/physiol.00030.2015View
Published (Version of record) Open Access

Abstract

Chronic musculoskeletal pain is debilitating and affects ∼ 20% of adults. Tissue acidosis is present in painful musculoskeletal diseases like rheumatoid arthritis. ASICs are located on skeletal muscle and joint nociceptors as well as on nonneuronal cells in the muscles and joints, where they mediate nociception. This review discusses the properties of different types of ASICs, factors affecting their pH sensitivity, and their role in musculoskeletal hyperalgesia and inflammation.
Nociception Signal Transduction Myositis - metabolism Humans Muscle, Skeletal - innervation Muscle, Skeletal - metabolism Animals Musculoskeletal Pain - metabolism Musculoskeletal Pain - physiopathology Pain Threshold Myositis - physiopathology Acid Sensing Ion Channels - metabolism Disease Models, Animal Hydrogen-Ion Concentration

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