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Actin and myosin dynamics are independent during Drosophila embryonic wound repair
Journal article   Open access   Peer reviewed

Actin and myosin dynamics are independent during Drosophila embryonic wound repair

Anna B. Kobb, Katheryn E. Rothenberg and Rodrigo Fernandez-Gonzalez
Molecular biology of the cell, Vol.30(23), pp.2901-2912
11/01/2019
DOI: 10.1091/mbc.E18-11-0703
PMCID: PMC6822589
PMID: 31553671
url
https://doi.org/10.1091/mbc.E18-11-0703View
Published (Version of record) Open Access

Abstract

Collective cell movements play a central role in embryonic development, tissue repair, and metastatic disease. Cell movements are often coordinated by supracellular networks formed by the cytoskeletal protein actin and the molecular motor nonmuscle myosin II. During wound closure in the embryonic epidermis, the cells around the wound migrate collectively into the damaged region. In Drosophila embryos, mechanical tension stabilizes myosin at the wound edge, facilitating the assembly of a supracellular myosin cable around the wound that coordinates cell migration. Here, we show that actin is also stabilized at the wound edge. However, loss of tension or myosin activity does not affect the dynamics of actin at the wound margin. Conversely, pharmacological stabilization of actin does not affect myosin levels or dynamics around the wound. Together, our data suggest that actin and myosin are independently regulated during embryonic wound closure, thus conferring robustness to the embryonic wound healing response.
Cell Biology Life Sciences & Biomedicine Science & Technology

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