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Activation of Crtc2/Creb1 in skeletal muscle enhances weight loss during intermittent fasting
Journal article   Peer reviewed

Activation of Crtc2/Creb1 in skeletal muscle enhances weight loss during intermittent fasting

Nelson E Bruno, Jerome C Nwachukwu, David C Hughes, Sathish Srinivasan, Richard Hawkins, David Sturgill, Gordon L Hager, Stephen Hurst, Shey-Shing Sheu, Sue C Bodine, …
The FASEB journal, Vol.35(12), pp.e21999-n/a
12/2021
DOI: 10.1096/fj.202100171R
PMCID: PMC10236530
PMID: 34748223

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Abstract

The Creb-Regulated Transcriptional Coactivator (Crtc) family of transcriptional coregulators drive Creb1-mediated transcription effects on metabolism in many tissues, but the in vivo effects of Crtc2/Creb1 transcription on skeletal muscle metabolism are not known. Skeletal muscle-specific overexpression of Crtc2 (Crtc2 mice) induced greater mitochondrial activity, metabolic flux capacity for both carbohydrates and fats, improved glucose tolerance and insulin sensitivity, and increased oxidative capacity, supported by upregulation of key metabolic genes. Crtc2 overexpression led to greater weight loss during alternate day fasting (ADF), selective loss of fat rather than lean mass, maintenance of higher energy expenditure during the fast and reduced binge-eating during the feeding period. ADF downregulated most of the mitochondrial electron transport genes, and other regulators of mitochondrial function, that were substantially reversed by Crtc2-driven transcription. Glucocorticoids acted with AMPK to drive atrophy and mitophagy, which was reversed by Crtc2/Creb1 signaling. Crtc2/Creb1-mediated signaling coordinates metabolic adaptations in skeletal muscle that explain how Crtc2/Creb1 regulates metabolism and weight loss.
Animals Cyclic AMP Response Element-Binding Protein - physiology Energy Metabolism Fasting Insulin Resistance Male Mice Mice, Transgenic Muscle, Skeletal - physiology Transcription Factors - physiology Weight Loss - physiology

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