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All-or-None Ca2+ Release from Intracellular Stores Triggered by Ca2+ Influx through Voltage-Gated Ca2+ Channels in Rat Sensory Neurons
Journal article   Open access   Peer reviewed

All-or-None Ca2+ Release from Intracellular Stores Triggered by Ca2+ Influx through Voltage-Gated Ca2+ Channels in Rat Sensory Neurons

Yuriy M Usachev and Stanley A Thayer
The Journal of neuroscience, Vol.17(19), pp.7404-7414
10/01/1997
DOI: 10.1523/JNEUROSCI.17-19-07404.1997
PMID: 9295386
url
https://doi.org/10.1523/JNEUROSCI.17-19-07404.1997View
Published (Version of record) Open Access

Abstract

Ca 2+ -induced Ca 2+ release (CICR) from intracellular stores amplifies the Ca 2+ signal that results from depolarization. In neurons, the amplification has been described as a graded process. Here we show that regenerative CICR develops as an all-or-none event in cultured rat dorsal root ganglion neurons in which ryanodine receptors have been sensitized to Ca 2+ by caffeine. We used indo-1-based microfluorimetry in combination with whole-cell patch-clamp recording to characterize the relationship between Ca 2+ influx and Ca 2+ release. Regenerative release of Ca 2+ was triggered when action potential-induced Ca 2+ influx increased the intracellular Ca 2+ concentration ([Ca 2+ ] i ) above threshold. The threshold was modulated by caffeine and intraluminal Ca 2+ . A relative refractory period followed CICR. The pharmacological profile of the response was consistent with Ca 2+ influx through voltage-gated Ca 2+ channels triggering release from ryanodine-sensitive stores. The activation of a suprathreshold response increased more than fivefold the amplitude and duration of the [Ca 2+ ] i transient. The switch to a suprathreshold response was regulated very precisely in that addition of a single action potential to the stimulus train was sufficient for this transformation. Confocal imaging experiments showed that CICR facilitated propagation of the Ca 2+ signal from the plasmalemma to the nucleus. This all-or-none reaction may serve as a switch that determines whether a given electrical signal will be transduced into a local or widespread increase in [Ca 2+ ] i .
intracellular Ca2 Ca2+ stores nucleoplasmic Ca2 voltage-gated Ca2+ channels dorsal root ganglion Ca2+-induced Ca2+release ryanodine receptors

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