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Alterations in the muscle force transfer apparatus in aged rats during unloading and reloading: impact of microRNA‐31
Journal article   Open access   Peer reviewed

Alterations in the muscle force transfer apparatus in aged rats during unloading and reloading: impact of microRNA‐31

David C. Hughes, George R. Marcotte, Leslie M. Baehr, Daniel W. D. West, Andrea G. Marshall, Scott M. Ebert, Arik Davidyan, Christopher M. Adams, Sue C Bodine and Keith Baar
The Journal of physiology, Vol.596(14), pp.2883-2900
07/14/2018
DOI: 10.1113/JP275833
PMCID: PMC6046073
PMID: 29726007
url
https://doi.org/10.1113/JP275833View
Published (Version of record) Open Access

Abstract

Force transfer is integral for maintaining skeletal muscle structure and function. One important component is dystrophin. There is limited understanding of how force transfer is impacted by age and loading. Here, we investigate the force transfer apparatus in muscles of adult and old rats exposed to periods of disuse and reloading. Our results demonstrate an increase in dystrophin protein during the reloading phase in the adult tibialis anterior muscle that is delayed in the old muscle. The consequence of this delay is an increased susceptibility towards contraction‐induced muscle injury. Central to the lack of dystrophin protein is an increase in miR‐31, a microRNA that inhibits dystrophin translation. In vivo electroporation with a miR‐31 sponge led to increased dystrophin protein and decreased contraction‐induced muscle injury in old skeletal muscle. Overall, our results detail the importance of the force transfer apparatus and provide new mechanisms for contraction‐induced injury in ageing skeletal muscle.
Muscle Research Paper

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