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Alveolar type I cells can give rise to KRAS-induced lung adenocarcinoma
Journal article   Open access   Peer reviewed

Alveolar type I cells can give rise to KRAS-induced lung adenocarcinoma

Minxiao Yang, Hua Shen, Per Flodby, Michael Koss, Rania Bassiouni, Yixin Liu, Tea Jashashvili, Aaron Neely, Ezuka Ogbolu, Jonathan Castillo, …
Cell reports, Vol.42(12), 113286
12/26/2023
DOI: 10.1016/j.celrep.2023.113286
PMCID: PMC10842735
PMID: 37995179
url
https://doi.org/10.1016/j.celrep.2023.113286View
Published (Version of record) Open Access

Abstract

Lung adenocarcinoma (LUAD) is the most prevalent subtype of lung cancer and presents clinically with a high degree of biological heterogeneity and distinct clinical outcomes. The current paradigm of LUAD etiology posits alveolar epithelial type II (AT2) cells as the primary cell of origin, while the role of AT1 cells in LUAD oncogenesis remains unknown. Here, we examine oncogenic transformation in mouse Gram-domain containing 2 (Gramd2)+ AT1 cells via oncogenic KRASG12D. Activation of KRASG12D in AT1 cells induces multifocal LUAD, primarily of papillary histology. Furthermore, KRT8+ intermediate cell states were observed in both AT2- and AT1-derived LUAD, but SCGB3A2+, another intermediate cell marker, was primarily associated with AT1 cells, suggesting different mechanisms of tumor evolution. Collectively, our study reveals that Gramd2+ AT1 cells can serve as a cell of origin for LUAD and suggests that distinct subtypes of LUAD based on cell of origin be considered in the development of therapeutics.
Lung Cancer Adenocarcinoma of Lung alveolar epithelial cells Animals cell of origin Cell Transformation CP: Cancer Kras(G12D) signaling KRT8(+) intermediate cell states Lung Neoplasms Mice Neoplastic oncogenesis Proto-Oncogene Proteins p21(ras) spatial transcriptomic sequencing

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