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An Alternative Form of Replication Protein A Prevents Viral Replication in Vitro
Journal article   Open access   Peer reviewed

An Alternative Form of Replication Protein A Prevents Viral Replication in Vitro

Aaron C Mason, Stuart J Haring, John M Pryor, Cathy A Staloch, Tze Fei Gan and Marc S Wold
The Journal of biological chemistry, Vol.284(8), pp.5324-5331
02/20/2009
DOI: 10.1074/jbc.M808963200
PMCID: PMC2643524
PMID: 19116208
url
https://doi.org/10.1074/jbc.M808963200View
Published (Version of record) Open Access

Abstract

Replication protein A (RPA), the eukaryotic single-stranded DNA-binding complex, is essential for multiple processes in cellular DNA metabolism. The “canonical” RPA is composed of three subunits (RPA1, RPA2, and RPA3); however, there is a human homolog to the RPA2 subunit, called RPA4, that can substitute for RPA2 in complex formation. We demonstrate that the resulting “alternative” RPA (aRPA) complex has solution and DNA binding properties indistinguishable from the canonical RPA complex; however, aRPA is unable to support DNA replication and inhibits canonical RPA function. Two regions of RPA4, the putative L34 loop and the C terminus, are responsible for inhibiting SV40 DNA replication. Given that aRPA inhibits canonical RPA function in vitro and is found in nonproliferative tissues, these studies indicate that RPA4 expression may prevent cellular proliferation via replication inhibition while playing a role in maintaining the viability of quiescent cells.
DNA Repair, Recombination, and Chromosome Dynamics

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