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An injectable microparticle formulation provides long-term inhibition of hypothalamic ERK1/2 activity and sympathetic excitation in rats with heart failure
Journal article   Peer reviewed

An injectable microparticle formulation provides long-term inhibition of hypothalamic ERK1/2 activity and sympathetic excitation in rats with heart failure

Youssef W Naguib, Yang Yu, Shun-Guang Wei, Angie Morris, Brittany E Givens, Aml I Mekkawy, Robert M Weiss, Robert B Felder and Aliasger K Salem
Molecular pharmaceutics, Vol.17(9), pp.3643-3648
09/08/2020
DOI: 10.1021/acs.molpharmaceut.0c00501
PMID: 32786958
url
https://www.ncbi.nlm.nih.gov/pmc/articles/7641013View
Open Access

Abstract

Sympathetic excitation contributes to clinical deterioration in systolic heart failure (HF). Significant inhibition of hypothalamic paraventricular nucleus (PVN) ERK1/2 signaling and a subsequent reduction of plasma norepinephrine (NE) levels in HF rats were achieved two weeks after a single subcutaneous injection of PD98059-loaded polymeric microparticles, without apparent adverse events, while blank microparticles had no effect. Similar reductions in plasma NE, a general indicator of sympathetic excitation, were previously achieved in HF rats by intracerebroventricular infusion of PD98059 or genetic knockdown of PVN ERK1/2 expression. This study presents a clinically feasible therapeutic approach to the central abnormalities contributing to HF progression.
heart failure 2 PD98059 p-ERK1 MEK1 2 inhibitor Microparticles hypothalamic paraventricular nucleus

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