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Autosomal recessive nonsyndromic hearing loss
Journal article   Peer reviewed

Autosomal recessive nonsyndromic hearing loss

Rachel A Sundstrom, Lut van Laer, Guy Van Camp and Richard J.H Smith
American journal of medical genetics, Vol.89(3), pp.123-129
09/24/1999
DOI: 10.1002/(SICI)1096-8628(19990924)89:3<123::AID-AJMG2>3.0.CO;2-P
PMID: 10704186

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Abstract

Nearly all genes for autosomal recessive nonsyndromal inherited hearing loss (ARNSHL) localized thus far cause prelingual severe to profound or profound hearing impairment. Of the 25 reported loci, most have been identified using single consanguineous families. Six of these genes have been cloned and encode a variety of proteins, including ion channels, extracellular matrix components, cytoskeletal components, and proteins essential for synaptic vesicular trafficking. One of these genes appears to be responsible for approximately 50% of all congenital severe to profound or profound hearing loss in many world populations, and mutations in two other genes can lead to either syndromic or nonsyndromic forms of deafness. The identification of additional genes that cause ARNSHL and elucidation of their function will refine our understanding of auditory physiology at the molecular level. Am. J. Med. Genet. (Semin. Med. Genet.) 89:123–129, 1999. © 2000 Wiley‐Liss, Inc.
MYO15 OTOF GJB2 MYO7A PDS TECTA

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