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BAG3 Attenuates Ischemia-Induced Skeletal Muscle Necroptosis in Diabetic Experimental Peripheral Artery Disease
Journal article   Open access   Peer reviewed

BAG3 Attenuates Ischemia-Induced Skeletal Muscle Necroptosis in Diabetic Experimental Peripheral Artery Disease

Arul Mani, Karthik Dhanabalan, Victor Lamin, Thomas Wong, Madhu Singh and Ayotunde Dokun
International journal of molecular sciences, Vol.23(18), p.10715
09/15/2022
DOI: 10.3390/ijms231810715
PMCID: PMC9502689
PMID: 36142618
url
https://doi.org/10.3390/ijms231810715View
Published (Version of record) Open Access

Abstract

Peripheral artery disease (PAD) is characterized by impaired blood flow to the lower extremities, resulting in ischemic limb injuries. Individuals with diabetes and PAD typically have more severe ischemic limb injuries and limb amputations, but the mechanisms involved are poorly understood. Previously, we identified BAG3 as a gene within a mouse genetic locus termed limb salvage QTL1 on mouse chromosome 7 that determined the extent of limb necrosis following ischemic injury in C57Bl/6 mice. Whether BAG3 deficiency plays a role in the severe ischemic injury observed in diabetic PAD is not known. In vitro, we found simulated ischemia enhanced BAG3 expression in primary human skeletal muscle cells, whereas BAG3 knockdown increased necroptosis markers and decreased cell viability. In vivo, ischemic skeletal muscles from hind limbs of high-fat diet (HFD)-fed mice showed poor BAG3 expression compared to normal chow diet (NCD)-fed mice, and this was associated with increased limb amputations. BAG3 overexpression in ischemic skeletal muscles from hind limbs of HFD mice rescued limb amputation and improved autophagy, necroptosis, skeletal muscle function and regeneration. Therefore, BAG3 deficiency in ischemic skeletal muscles contributes to the severity of ischemic limb injury in diabetic PAD, likely through autophagy and necroptosis pathways.
Apoptosis Blood Flow Diabetes Ischemia Musculoskeletal System Vein & Artery Diseases Amputation Autophagy Cell viability Chromosome 7 Diabetes mellitus Diet High fat diet Injuries Limbs Muscles Necroptosis Necrosis Phagocytosis Protein expression Proteins Skeletal muscle Vascular diseases

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