Logo image
Bisphenol F exposure in adolescent heterogeneous stock (HS) rats affects growth and adiposity
Journal article   Open access   Peer reviewed

Bisphenol F exposure in adolescent heterogeneous stock (HS) rats affects growth and adiposity

Valerie A Wagner, Karen C Clark, Leslie Carrillo-Sáenz, Katie A Holl, Miriam Velez-Bermudez, Derek Simonsen, Justin L Grobe, Kai Wang, Andrew Thurman, Leah C Solberg Woods, …
Toxicological sciences, Vol.181(2), pp.246-261
03/23/2021
DOI: 10.1093/toxsci/kfab035
PMID: 33755180
url
https://doi.org/10.1093/toxsci/kfab035View
Published (Version of record) Open Access

Abstract

Abstract Bisphenol F (BPF) is increasingly substituting bisphenol A (BPA) in manufacturing polycarbonates and consumer products. The cardiometabolic effects of BPF in either humans or model organisms are not clear, and no studies to date have investigated the role of genetic background on susceptibility to BPF-induced cardiometabolic traits. The primary goal of this project was to determine if BPF exposure influences growth and adiposity in male N: NIH Heterogeneous Stock (HS) rats, a genetically heterogeneous population. Littermate pairs of male HS rats were randomly exposed to either vehicle (0.1% Ethanol) or 1.125 µg/ml BPF in 0.1% Ethanol for five weeks in drinking water starting at three weeks-of-age. Water consumption and body weight was measured weekly, body composition was determined using nuclear magnetic resonance (NMR), urine and feces were collected in metabolic cages, and blood and tissues were collected at the end of the study. BPF-exposed rats showed significantly increased body growth and abdominal adiposity, risk factors for cardiometabolic disease. Urine output was increased in BPF-exposed rats, driving a trend in increased creatinine clearance. We also report the first relationship between a bisphenol metabolizing enzyme and a bisphenol-induced phenotype. Preliminary heritability estimates of significant phenotypes suggest that BPF exposure may alter trait variation. These findings support BPF exposure as a cardiometabolic disease risk factor and indicate that the HS rat will be a useful model for dissecting gene by BPF interactions on metabolic health.

Details

Metrics

Logo image