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CDC14A phosphatase is essential for hearing and male fertility in mouse and human
Journal article   Open access   Peer reviewed

CDC14A phosphatase is essential for hearing and male fertility in mouse and human

Hela Azaiez, Ayesha Imtiaz, Inna A Belyantseva, Kevin T Booth, Alisha J Beirl, Elizabeth A Wilson, Richard J H Smith, Cristina Fenollar-Ferrer, Rasheeda Bashir, Ihtisham Bukhari, …
Human molecular genetics, Vol.27(5), pp.780-798
03/01/2018
DOI: 10.1093/hmg/ddx440
PMCID: PMC6059191
PMID: 29293958
url
https://doi.org/10.1093/hmg/ddx440View
Published (Version of record) Open Access

Abstract

The Cell Division-Cycle-14 gene encodes a dual-specificity phosphatase necessary in yeast for exit from mitosis. Numerous disparate roles of vertebrate Cell Division-Cycle-14 (CDC14A) have been proposed largely based on studies of cultured cancer cells in vitro. The in vivo functions of vertebrate CDC14A are largely unknown. We generated and analyzed mutations of zebrafish and mouse CDC14A, developed a computational structural model of human CDC14A protein and report four novel truncating and three missense alleles of CDC14A in human families segregating progressive, moderate-to-profound deafness. In five of these families segregating pathogenic variants of CDC14A, deaf males are infertile, while deaf females are fertile. Several recessive mutations of mouse Cdc14a, including a CRISPR/Cas9-edited phosphatase-dead p.C278S substitution, result in substantial perinatal lethality, but survivors recapitulate the human phenotype of deafness and male infertility. CDC14A protein localizes to inner ear hair cell kinocilia, basal bodies and sound-transducing stereocilia. Auditory hair cells of postnatal Cdc14a mutants develop normally, but subsequently degenerate causing deafness. Kinocilia of germ-line mutants of mouse and zebrafish have normal lengths, which does not recapitulate the published cdc14aa knockdown morphant phenotype of short kinocilia. In mutant male mice, degeneration of seminiferous tubules and spermiation defects result in low sperm count, and abnormal sperm motility and morphology. These findings for the first time define a new monogenic syndrome of deafness and male infertility revealing an absolute requirement in vivo of vertebrate CDC14A phosphatase activity for hearing and male fertility.
Phosphoric Monoester Hydrolases - genetics Genetic Association Studies Hearing Loss - physiopathology Zebrafish Proteins - metabolism Humans Male Protein Tyrosine Phosphatases - metabolism Zebrafish - embryology Zebrafish - genetics Hearing Loss - genetics Protein Tyrosine Phosphatases - genetics Animals Infertility, Male - genetics Mice, Mutant Strains Pedigree CRISPR-Cas Systems Female Testis - physiopathology Zebrafish Proteins - genetics Phosphoric Monoester Hydrolases - chemistry

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