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Cdc42 Regulates Microtubule-Dependent Golgi Positioning
Journal article   Open access

Cdc42 Regulates Microtubule-Dependent Golgi Positioning

Heidi Hehnly, Weidong Xu, Ji-Long Chen and Mark Stamnes
Traffic (Copenhagen, Denmark), Vol.11(8), pp.1067-1078
08/01/2010
DOI: 10.1111/j.1600-0854.2010.01082.x
PMCID: PMC2904418
PMID: 20525016
url
https://doi.org/10.1111/j.1600-0854.2010.01082.xView
Published (Version of record) Open Access

Abstract

The molecular mechanisms underlying cytoskeleton-dependent Golgi positioning are poorly understood. In mammalian cells, the Golgi apparatus is localized near the juxtanuclear centrosome via dynein-mediated motility along microtubules. Previous studies implicate Cdc42 in regulating dynein-dependent motility. Here we show that reduced expression of the Cdc42-specific GTPase-activating protein, ARHGAP21, inhibits the ability of dispersed Golgi membranes to reposition at the centrosome following nocodazole treatment and washout. Cdc42 regulation of Golgi positioning appears to involve ARF1 and a binding interaction with the vesicle-coat protein coatomer. We tested whether Cdc42 directly affects motility, as opposed to the formation of a trafficking intermediate, using a Golgi capture and motility assay in permeabilized cells. Disrupting Cdc42 activation or the coatomer/Cdc42 binding interaction stimulated Golgi motility. The coatomer/Cdc42-sensitive motility was blocked by the addition of an inhibitory dynein antibody. Together, our results reveal that dynein and microtubule-dependent Golgi positioning is regulated by ARF1-, coatomer-, and ARHGAP21-dependent Cdc42 signaling.
Cell Biology Life Sciences & Biomedicine Science & Technology

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