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Characterization of Type I Interferon-Associated Chemokines and Cytokines in Lacrimal Glands of Nonobese Diabetic Mice
Journal article   Open access   Peer reviewed

Characterization of Type I Interferon-Associated Chemokines and Cytokines in Lacrimal Glands of Nonobese Diabetic Mice

Merri-Grace Allred, Michael S. Chimenti, Ashley E. Ciecko, Yi-Guang Chen and Scott M. Lieberman
International journal of molecular sciences, Vol.22(7), p.3767
04/01/2021
DOI: 10.3390/ijms22073767
PMCID: PMC8038628
PMID: 33916486
url
https://doi.org/10.3390/ijms22073767View
Published (Version of record) Open Access

Abstract

Type I interferons (IFNs) are required for spontaneous lacrimal gland inflammation in the nonobese diabetic (NOD) mouse model of Sjogren's disease, but the consequences of type I IFN signaling are not well-defined. Here, we use RNA sequencing to define cytokine and chemokine genes upregulated in lacrimal glands of NOD mice in a type I IFN-dependent manner. Interleukin (IL)-21 was the highest differentially expressed cytokine gene, and Il21 knockout NOD mice were relatively protected from lacrimal gland inflammation. We defined a set of chemokines upregulated early in disease including Cxcl9 and Cxcl10, which share a receptor, CXCR3. CXCR3(+) T cells were enriched in lacrimal glands with a dominant proportion of CXCR3(+) regulatory T cells. Together these data define the early cytokine and chemokine signals associated with type I IFN-signaling in the development of lacrimal gland inflammation in NOD mice providing insight into the role of type I IFN in autoimmunity development.
Biochemistry & Molecular Biology Chemistry Chemistry, Multidisciplinary Life Sciences & Biomedicine Physical Sciences Science & Technology

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