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De novo variants of NR4A2 are associated with neurodevelopmental disorder and epilepsy
Journal article   Open access   Peer reviewed

De novo variants of NR4A2 are associated with neurodevelopmental disorder and epilepsy

Sakshi Singh, Aditi Gupta, Michael Zech, Ashley N Sigafoos, Karl J Clark, Yasemin Dincer, Matias Wagner, Jennifer B Humberson, Sarah Green, Koen van Gassen, …
Genetics in medicine, Vol.22(8), pp.1413-1417
08/2020
DOI: 10.1038/s41436-020-0815-4
PMCID: PMC7394879
PMID: 32366965
url
https://doi.org/10.1038/s41436-020-0815-4View
Published (Version of record) Open Access

Abstract

This study characterizes the clinical and genetic features of nine unrelated patients with de novo variants in the NR4A2 gene. Variants were identified and de novo origins were confirmed through trio exome sequencing in all but one patient. Targeted RNA sequencing was performed for one variant to confirm its splicing effect. Independent discoveries were shared through GeneMatcher. Missense and loss-of-function variants in NR4A2 were identified in patients from eight unrelated families. One patient carried a larger deletion including adjacent genes. The cases presented with developmental delay, hypotonia (six cases), and epilepsy (six cases). De novo status was confirmed for eight patients. One variant was demonstrated to affect splicing and result in expression of abnormal transcripts likely subject to nonsense-mediated decay. Our study underscores the importance of NR4A2 as a disease gene for neurodevelopmental disorders and epilepsy. The identified variants are likely causative of the seizures and additional developmental phenotypes in these patients.
Epilepsy - genetics Humans Intellectual Disability - genetics Muscle Hypotonia Neurodevelopmental Disorders - genetics Nuclear Receptor Subfamily 4, Group A, Member 2 Phenotype Whole Exome Sequencing

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