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Effect of an intravitreal antisense oligonucleotide on vision in Leber congenital amaurosis due to a photoreceptor cilium defect
Journal article

Effect of an intravitreal antisense oligonucleotide on vision in Leber congenital amaurosis due to a photoreceptor cilium defect

Artur V Cideciyan, Samuel G Jacobson, Arlene V Drack, Allen C Ho, Jason Charng, Alexandra V Garafalo, Alejandro J Roman, Alexander Sumaroka, Ian C Han, Maria D Hochstedler, …
Nature Medicine, Vol.25(2), pp.225-228
02/01/2019
DOI: 10.1038/s41591-018-0295-0
PMID: 30559420

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Abstract

Photoreceptor ciliopathies constitute the most common molecular mechanism of the childhood blindness Leber congenital amaurosis. Ten patients with Leber congenital amaurosis carrying the c.2991+1655A>G allele in the ciliopathy gene centrosomal protein 290 (CEP290) were treated (ClinicalTrials. gov no. NCT03140969) with intravitreal injections of an antisense oligonucleotide to restore correct splicing. There were no serious adverse events, and vision improved at 3 months. The visual acuity of one exceptional responder improved from light perception to 20/400.
Blindness Cell Biology Vision Visual Perception MEDICINE, RESEARCH & EXPERIMENTAL GENE-THERAPY BIOCHEMISTRY & MOLECULAR BIOLOGY Antigens, Neoplasm - genetics Cilia - pathology Intravitreal Injections Leber Congenital Amaurosis - physiopathology Humans Male Leber Congenital Amaurosis - drug therapy Young Adult Vision, Ocular Oligonucleotides, Antisense - therapeutic use Alleles Cilia - drug effects Adult Female Oligonucleotides, Antisense - administration & dosage Neoplasm Proteins - genetics Photoreceptor Cells, Vertebrate - pathology Antisense drugs Analysis Photoreceptors Genetic aspects Dosage and administration Research Children Drug therapy Health aspects Proteins Splicing Antisense oligonucleotides Visual acuity Acuity

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