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Elevated AF1q expression is a poor prognostic marker for adult acute myeloid leukemia patients with normal cytogenetics
Journal article   Open access   Peer reviewed

Elevated AF1q expression is a poor prognostic marker for adult acute myeloid leukemia patients with normal cytogenetics

Crawford J Strunk, Uwe Platzbecker, Christian Thiede, Markus Schaich, Thomas Illmer, Zizhen Kang, Patrick Leahy, Chunbiao Li, Xiuyan Xie, Mary J Laughlin, …
American journal of hematology, Vol.84(5), pp.308-309
05/2009
DOI: 10.1002/ajh.21396
PMID: 19396856
url
https://doi.org/10.1002/ajh.21396View
Published (Version of record) Open Access

Abstract

Nearly half of the patients with newly diagnosed acute myeloid leukemia have normal cytogenetics (NC-AML) and are classified as intermediate risk, but their 5-year overall survival (OS) ranges from 24 to 42%. Therefore, molecular biomarkers to identify poor-risk patients are needed. Elevated AF1q expression in the absence of specific poor cytogenetics is associated with poor outcomes in pediatric patients with AML and adult patients with myelodysplastic syndrome. We examined AF1q expression in 290 patients with NC-AML. We found that patients with low AF1q (n = 73) expression (AF1q(low)) have better OS (P = 0.026), disease-free survival (P = 0.1), and complete remission rate (P = 0.06) when compared with patients with high AF1q expression (AF1q(high) n = 217). The patients with AF1q(high) had significantly greater incidence of concurrent tyrosine kinase3 internal tandem duplication. A subgroup of the patients with AF1q(high) who received allogeneic stem cell transplantation (SCT) had a significant better relapse-free survival when compared with patients who received chemotherapy/autologous SCT (P = 0.04). This study suggests that high AF1q expression is a poor prognostic marker for adult patients with NC-AML.
Adolescent Adult Biomarkers, Tumor Blood Proteins - analysis Blood Proteins - metabolism Combined Modality Therapy Cytogenetic Analysis Disease-Free Survival Female fms-Like Tyrosine Kinase 3 - genetics Humans Leukemia, Myeloid, Acute - genetics Leukemia, Myeloid, Acute - metabolism Leukemia, Myeloid, Acute - pathology Leukemia, Myeloid, Acute - therapy Male Middle Aged Neoplasm Proteins - analysis Neoplasm Proteins - metabolism Prognosis Proto-Oncogene Proteins Stem Cell Transplantation Transplantation, Autologous Transplantation, Homologous Young Adult

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