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Fascin regulates protrusions and delamination to mediate invasive, collective cell migration in vivo
Journal article   Peer reviewed

Fascin regulates protrusions and delamination to mediate invasive, collective cell migration in vivo

Maureen C. Lamb, Kelsey K. Anliker and Tina L. Tootle
Developmental dynamics, Vol.249(8), pp.961-982
08/2020
DOI: 10.1002/dvdy.186
PMCID: PMC8900984
PMID: 32352613
url
https://www.ncbi.nlm.nih.gov/pmc/articles/8900984View
Open Access

Abstract

Background The actin bundling protein Fascin is essential for developmental cell migrations and promotes cancer metastasis. In addition to bundling actin, Fascin has several actin‐independent roles; how these other functions contribute to cell migration remains unclear. Border cell migration during Drosophila oogenesis provides an excellent model to study Fascin's various roles during invasive, collective cell migration. Results On‐time border cell migration during Stage 9 requires Fascin (Drosophila Singed). Fascin functions not only within the migrating border cells, but also within the nurse cells, the substrate for this migration. Fascin genetically interacts with the actin elongation factor Enabled to promote on‐time Stage 9 migration and overexpression of Enabled suppresses the defects seen with loss of Fascin. Loss of Fascin results in increased, shorter and mislocalized protrusions during migration. Additionally, loss of Fascin inhibits border cell delamination and increases E‐Cadherin (Drosophila Shotgun) adhesions on both the border cells and nurse cells. Conclusions Overall, Fascin promotes on‐time border cell migration during Stage 9 and contributes to multiple aspects of this invasive, collective cell migration, including both protrusion dynamics and delamination. These findings have implications beyond Drosophila, as border cell migration has emerged as a model to study mechanisms mediating cancer metastasis. Key Findings Fascin functions in the migrating border cells and their substrate to regulate on‐time border cell migration during Stage 9 of Drosophila oogenesis. Fascin genetically interacts with Enabled and regulates protrusion dynamics to promote migration. Fascin limits E‐Cadherin membrane localization on the border cells and surrounding nurse cells, and regulates delamination.
border cell Drosophila Enabled E‐Cadherin Fascin migration

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