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GENOME-WIDE ASSOCIATION STUDY OF BIPOLAR DISORDER IN EUROPEAN AMERICAN AND AFRICAN AMERICAN INDIVIDUALS
Journal article   Peer reviewed

GENOME-WIDE ASSOCIATION STUDY OF BIPOLAR DISORDER IN EUROPEAN AMERICAN AND AFRICAN AMERICAN INDIVIDUALS

Erin N Smith, Cinnamon S Bloss, Judith A Badner, Thomas Barrett, Pamela L Belmonte, Wade Berrettini, William Byerley, William Coryell, David Craig, Howard J Edenberg, …
Molecular psychiatry, Vol.14(8), pp.755-763
08/2009
DOI: 10.1038/mp.2009.43
PMCID: PMC3035981
PMID: 19488044
url
https://www.ncbi.nlm.nih.gov/pmc/articles/3035981View
Open Access

Abstract

To identify Bipolar Disorder (BD) genetic susceptibility factors, we conducted two genome-wide association (GWA) studies: one involving a sample of individuals of European ancestry (EA; n = 1,001 cases; n = 1,033 controls) and one involving a sample of individuals of African ancestry (AA; n = 345 cases; n = 670 controls). For the EA sample, SNPs with strongest statistical evidence for association included rs5907577 in an intergenic region at Xq27.1 (p = 1.6 × 10 -6 ) and rs10193871 in NAP5 at 2q21.2 (p = 9.8 × 10 -6 ). For the AA sample, SNPs with strongest statistical evidence for association included rs2111504 in DPY19L3 at 19q13.11 (p = 1.5 × 10 -6 ) and rs2769605 in NTRK2 at 9q21.33 (p = 4.5 × 10 -5 ). We also investigated whether we could provide support for three regions previously associated with BD, and we show that the ANK3 region replicates in our sample, along with some support for C15Orf53 ; other evidence implicates BD candidate genes such as SLITRK2 . We also tested the hypothesis that BD susceptibility variants exhibit genetic background-dependent effects; SNPs with the strongest statistical evidence for this included rs11208285 in ROR1 at 1p31.3 (p = 1.4 × 10 -6 ), rs4657247 in RGS5 at 1q23.3 (p = 4.1 × 10 -6 ), and rs7078071 in BTBD16 at 10q26.13 (p = 4.5 × 10 -6 ). This study is the first to conduct GWA of BD in individuals of AA and suggests that genetic variations that contribute to BD may vary as a function of ancestry.
Bipolar Genome Study NTRK2 DPY19L3 SLITRK2 C15Orf53 genetic background Genetic Information Association Network (GAIN) ROR1 ANK3 NAP5 allelic heterogeneity

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