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HIV-1 is dependent on its immature lattice to recruit IP6 for mature capsid assembly
Journal article   Peer reviewed

HIV-1 is dependent on its immature lattice to recruit IP6 for mature capsid assembly

Nadine Renner, Alex Kleinpeter, Donna L. Mallery, Anna Albecka, Rifat K. M. Faysal, Till Bocking, Adolfo Saiardi, Eric O. Freed and Leo C. James
Nature structural & molecular biology, Vol.30(3), pp.370-382
03/2023
DOI: 10.1038/s41594-022-00887-4
PMCID: PMC7614341
PMID: 36624347
url
https://discovery.ucl.ac.uk/10163385/1/Combined_Revised_NSMB%20manuscript%20Saiardi.pdfView
Open Access

Abstract

HIV-1 Gag metamorphoses inside each virion, from an immature lattice that forms during viral production to a mature capsid that drives infection. Here we show that the immature lattice is required to concentrate the cellular metabolite inositol hexakisphosphate (IP6) into virions to catalyze mature capsid assembly. Disabling the ability of HIV-1 to enrich IP6 does not prevent immature lattice formation or production of the virus. However, without sufficient IP6 molecules inside each virion, HIV-1 can no longer build a stable capsid and fails to become infectious. IP6 cannot be replaced by other inositol phosphate (IP) molecules, as substitution with other IPs profoundly slows mature assembly kinetics and results in virions with gross morphological defects. Our results demonstrate that while HIV-1 can become independent of IP6 for immature assembly, it remains dependent upon the metabolite for mature capsid formation.
Biophysics Cell Biology Biochemistry & Molecular Biology Life Sciences & Biomedicine Science & Technology

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