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High Frequency of Virus-Specific Interleukin-2-Producing CD4+ T Cells and Th1 Dominance during Lymphocytic Choriomeningitis Virus Infection
Journal article   Open access   Peer reviewed

High Frequency of Virus-Specific Interleukin-2-Producing CD4+ T Cells and Th1 Dominance during Lymphocytic Choriomeningitis Virus Infection

Steven M Varga and Raymond M Welsh
Journal of virology, Vol.74(9), pp.4429-4432
Note
05/2000
DOI: 10.1128/JVI.74.9.4429-4432.2000
PMCID: PMC111961
PMID: 10756059
url
https://doi.org/10.1128/JVI.74.9.4429-4432.2000View
Published (Version of record) Open Access

Abstract

Analysis of C57BL/6 mice acutely infected with lymphocytic choriomeningitis virus (LCMV) by using intracellular cytokine staining revealed a high frequency (2 to 10%) of CD4 + T cells secreting the Th1-associated cytokines interleukin-2 (IL-2), gamma interferon (IFN-γ), and tumor necrosis factor alpha, with no concomitant increase in the frequency of CD4 + T cells secreting the Th2-associated cytokines IL-4, IL-5, and IL-10 following stimulation with viral peptides. In LCMV-infected C57BL/6 CD8 −/− mice, more than 20% of the CD4 + T cells secreted IFN-γ after viral peptide stimulation, whereas less than 1% of the CD4 + T cells secreted IL-4 under these same conditions. Mice persistently infected with a high dose of LCMV clone 13 also generated a virtually exclusive Th1 response. Thus, LCMV induces a much more profound virus-specific CD4 + T-cell response than previously recognized, and it is dramatically skewed to a Th1 phenotype.
Pathogenesis and Immunity

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