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IFN-γ-mediated suppression of coronavirus replication in glial-committed progenitor cells
Journal article   Open access   Peer reviewed

IFN-γ-mediated suppression of coronavirus replication in glial-committed progenitor cells

Lucia Whitman, Haixia Zhou, Stanley Perlman and Thomas E Lane
Virology, Vol.384(1), pp.209-215
2009
DOI: 10.1016/j.virol.2008.10.036
PMCID: PMC2779567
PMID: 19059617
url
https://doi.org/10.1016/j.virol.2008.10.036View
Published (Version of record) Open Access

Abstract

The neurotropic JHM strain of mouse hepatitis virus (JHMV) replicates primarily within glial cells following intracranial inoculation of susceptible mice, with relative sparing of neurons. This study demonstrates that glial cells derived from neural progenitor cells are susceptible to JHMV infection and that treatment of infected cells with IFN-γ inhibits viral replication in a dose-dependent manner. Although type I IFN production is muted in JHMV-infected glial cultures, IFN-β is produced following IFN-γ-treatment of JHMV-infected cells. Also, direct treatment of infected glial cultures with recombinant mouse IFN-α or IFN-β inhibits viral replication. IFN-γ-mediated control of JHMV replication is dampened in glial cultures derived from the neural progenitor cells of type I receptor knock-out mice. These data indicate that JHMV is capable of infecting glial cells generated from neural progenitor cells and that IFN-γ-mediated control of viral replication is dependent, in part, on type I IFN secretion.
Virus Neural progenitor cells Interferons Central nervous system

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