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Inhibition of hepatic gluconeogenesis and enhanced glucose uptake contribute to the development of hypoglycemia in mice bearing interleukin-1β-secreting tumor
Journal article   Open access   Peer reviewed

Inhibition of hepatic gluconeogenesis and enhanced glucose uptake contribute to the development of hypoglycemia in mice bearing interleukin-1β-secreting tumor

Shulamit Metzger, Samir Nusair, David Planer, Varda Barash, Orit Pappo, Joel Shilyansky and Tova CHAJEK-SHAUL
Endocrinology (Philadelphia), Vol.145(11), pp.5150-5156
2004
DOI: 10.1210/en.2004-0323
PMID: 15297440
url
https://doi.org/10.1210/en.2004-0323View
Published (Version of record) Open Access

Abstract

Mice bearing IL-1β-secreting tumor were used to study the chronic effect of IL-1β on glucose metabolism. Mice were injected with syngeneic tumor cells transduced with the human IL-1β gene. Serum IL-1β levels increased exponentially with time. Secretion of IL-1β from the developed tumors was associated with decreased food consumption, reduced body weight, and reduced blood glucose levels. Body composition analysis revealed that IL-1β caused a significant loss in fat tissue without affecting lean body mass and water content. Hepatic phosphoenolpyruvate carboxykinase and glucose-6-phosphatase activities and mRNA levels of these enzymes were reduced, and 2-deoxy-glucose uptake by peripheral tissues was enhanced. mRNA levels of glucose transporters (Gluts) in the liver were determined by real-time PCR analysis. Glut-3 mRNA levels were up-regulated by IL-1β. Glut-1 and Glut-4 mRNA levels in IL-1β mice were similar to mRNA levels in pair-fed mice bearing nonsecreting tumor. mRNA level of Glut-2, the major Glut of the liver, was down-regulated by IL-1β. We concluded that both decreased glucose production by the liver and enhanced glucose disposal lead to the development of hypoglycemic in mice bearing IL-1β-secreting tumor. The observed changes in expression of hepatic Gluts that are not dependent on insulin may contribute to the increased glucose uptake.
Biological and medical sciences Fundamental and applied biological sciences. Psychology Vertebrates: endocrinology

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