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Interaction of the tetratricopeptide repeat domain of aryl hydrocarbon receptor-interacting protein-like 1 with the regulatory Pγ subunit of phosphodiesterase 6
Journal article   Open access   Peer reviewed

Interaction of the tetratricopeptide repeat domain of aryl hydrocarbon receptor-interacting protein-like 1 with the regulatory Pγ subunit of phosphodiesterase 6

Ravi P Yadav, Kimberly Boyd, Liping Yu and Nikolai O Artemyev
The Journal of biological chemistry, Vol.294(43), pp.15795-15807
10/25/2019
DOI: 10.1074/jbc.RA119.010666
PMCID: PMC6816093
PMID: 31488544
url
https://doi.org/10.1074/jbc.RA119.010666View
Published (Version of record) Open Access

Abstract

Phosphodiesterase-6 (PDE6) is key to both phototransduction and health of rods and cones. Proper folding of PDE6 relies on the chaperone activity of aryl hydrocarbon receptor-interacting protein-like 1 (AIPL1), and mutations in both PDE6 and AIPL1 can cause a severe form of blindness. Although AIPL1 and PDE6 are known to interact via the FK506-binding protein domain of AIPL1, the contribution of the tetratricopeptide repeat (TPR) domain of AIPL1 to its chaperone function is poorly understood. Here, we demonstrate that AIPL1-TPR interacts specifically with the regulatory Pγ subunit of PDE6. Use of NMR chemical shift perturbation (CSP) mapping technique revealed the interface between the C-terminal portion of Pγ and AIPL1-TPR. Our solution of the crystal structure of the AIPL1-TPR domain provided additional information, which together with the CSP data enabled us to generate a model of this interface. Biochemical analysis of chimeric AIPL1-AIP proteins supported this model and also revealed a correlation between the affinity of AIPL1-TPR for Pγ and the ability of Pγ to potentiate the chaperone activity of AIPL1. Based on these results, we present a model of the larger AIPL1-PDE6 complex. This supports the importance of simultaneous interactions of AIPL1-FK506-binding protein with the prenyl moieties of PDE6 and AIPL1-TPR with the Pγ subunit during the folding and/or assembly of PDE6. This study sheds new light on the versatility of TPR domains in protein folding by describing a novel TPR-protein binding partner, Pγ, and revealing that this subunit imparts AIPL1 selectivity for its client.
Adaptor Proteins, Signal Transducing - chemistry Cyclic Nucleotide Phosphodiesterases, Type 6 - metabolism Temperature Magnetic Resonance Spectroscopy Protein Structure, Secondary Tacrolimus Binding Proteins - chemistry Humans Models, Molecular Tacrolimus Binding Proteins - metabolism Protein Subunits - metabolism Animals Tetratricopeptide Repeat HEK293 Cells Protein Denaturation Protein Binding Mice Kinetics Adaptor Proteins, Signal Transducing - metabolism

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