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Lmx1a is essential for marginal cell differentiation and stria vascularis formation
Journal article   Open access   Peer reviewed

Lmx1a is essential for marginal cell differentiation and stria vascularis formation

Justine M. Renauld, Igor Y. Iskusnykh, Ebenezer N. Yamoah, Richard J. H. Smith, Corentin Affortit, David Z. He, Huizhan Liu, David Nichols, Judith Bouma, Mahesh K. Nayak, …
Frontiers in cell and developmental biology, Vol.13, 1537505
03/05/2025
DOI: 10.3389/fcell.2025.1537505
PMCID: PMC11920146
PMID: 40109362
url
https://doi.org/10.3389/fcell.2025.1537505View
Published (Version of record) Open Access

Abstract

The transcription factor Lmx1a is widely expressed during early inner ear development, and mice lacking Lmx1a expression exhibit fusion of cochlear and vestibular hair cells and fail to form the ductus reuniens and the endolymphatic sac. Lmx1a dreher ( Lmx1a dr/dr ), a recessive null mutation, results in non-functional Lmx1a expression, which expands from the outer sulcus to the stria vascularis and Reissner’s membrane. In the absence of Lmx1a , we observe a lack of proteins specific to the stria vascularis, such as BSND and KCNQ1 in marginal cells and CD44 in intermediate cells. Further analysis of the superficial epithelial cell layer at the expected stria vascularis location shows that the future intermediate cells migrate during embryonic development but subsequently disappear. Using antibodies against pendrin ( Slc26a4 ) in Lmx1a knockout (KO) mice, we observe an expansion of pendrin expression across the stria vascularis and Reissner’s membrane. Moreover, in the absence of Lmx1a expression, no endocochlear potential is observed. These findings highlight the critical role of Lmx1a in inner ear development, particularly in the differentiation of cochlear and vestibular structures, the recruitment of pigment cells, and the expression of proteins essential for hearing and balance.
<italic>Lmx1a</italic> pendrin Slc26a4 stria vascularis spiral prominence pigment cells

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