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Mast cell degranulation breaks peripheral tolerance
Journal article   Open access   Peer reviewed

Mast cell degranulation breaks peripheral tolerance

Victor C de Vries, Anna Wasiuk, Kathryn A Bennett, Micah J Benson, Raul Elgueta, Thomas J Waldschmidt and Randolph. J Noelle
American journal of transplantation, Vol.9(10), pp.2270-2280
10/2009
DOI: 10.1111/j.1600-6143.2009.02755.x
PMCID: PMC3808998
PMID: 19681828
url
https://doi.org/10.1111/j.1600-6143.2009.02755.xView
Published (Version of record) Open Access

Abstract

Mast cells (MC) have been shown to mediate regulatory T-cell (T reg ) dependent, peripheral allograft tolerance in both skin and cardiac transplants. Furthermore, T reg have been implicated in mitigating IgE mediated MC degranulation, establishing a dynamic, reciprocal relationship between MC and T reg in controlling inflammation. In an allograft tolerance model, it is now shown that intragraft or systemic MC degranulation results in the transient loss of T reg suppressor activities with the acute, T-cell dependent rejection of established, tolerant allografts. Upon degranulation, MC mediators can be found in the skin, T reg rapidly leave the graft, MC accumulate in the regional lymph node and the T reg are impaired in the expression of suppressor molecules. Such a dramatic reversal of T reg function and tissue distribution by MC degranulation underscores how allergy may causes the transient breakdown of peripheral tolerance and episodes of acute T-cell inflammation.

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