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MicroRNAs and Polycystic Kidney Disease
Journal article   Peer reviewed

MicroRNAs and Polycystic Kidney Disease

Lama Noureddine, Sachin Hajarnis and Vishal Patel
Drug discovery today. Disease models, Vol.10(3), pp.e137-e143
2013
DOI: 10.1016/j.ddmod.2013.10.001
PMCID: PMC4159181
PMID: 25221607
url
http://doi.org/10.1016/j.ddmod.2013.10.001View
Open Access

Abstract

Polycystic kidney disease (PKD), the most common genetic cause of chronic renal failure, is characterized by the presence of numerous fluid-filled cysts in renal parenchyma. Despite recent progress, no FDA-approved therapy is available to retard cyst growth. Here, we review current evidence implicating two groups of miRNAs - the miR-17~92 cluster and miR-200s - in the pathogenesis of PKD. We present a new hypothesis for cyst growth involving miRNAs and regulation of PKD gene dosage. We propose that manipulating miRNA function in an attempt to normalize PKD gene dosage represents a novel therapeutic strategy in PKD.

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