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Modulation of Wnt/[beta]-catenin signaling in IL-17A-mediated macrophage polarization of RAW264.7 cells
Journal article   Open access   Peer reviewed

Modulation of Wnt/[beta]-catenin signaling in IL-17A-mediated macrophage polarization of RAW264.7 cells

Jing Xue, Jia Ma, Chao Yuan, Jiali Yang, Fuyang Song, Xiaoming Liu and Dandan Yang
Brazilian journal of medical and biological research, Vol.53(8), e9488
08/01/2020
DOI: 10.1590/1414-431X20209488
PMCID: PMC7307890
PMID: 32578719
url
https://doi.org/10.1590/1414-431X20209488View
Published (Version of record) Open Access

Abstract

Macrophages play pivotal roles in host defense and immune homeostasis, which have two major functional polarization states, the classically activated M1 and the alternatively activated M2. Interleukin (IL)-17A is an immune modulator able to shape macrophage phenotypes. Wnt/[beta]-catenin is a developmental signaling pathway that plays crucial roles in morphogenesis and tissue homeostasis, which has also been recently demonstrated playing roles in immune regulation. A growing amount of evidence suggests that both Wnt and IL-17A signaling are involved in macrophage polarization. However, their interaction in macrophage polarization remains elusive. The aim of present study was to explore impacts of Wnt/[beta]-catenin on IL-17A-mediated macrophage M1/M2 polarization in murine monocyte/macrophage-like cell line RAW264.7. Results revealed that IL17A activated Wnt/[beta]-catenin signaling and induced macrophage M1 polarization, but inhibited M2 polarization. In contrast, the activation of Wnt/[beta]-catenin signaling led to the inhibition of M1 macrophage polarization but the promotion of M2 polarization. Importantly, the activation of Wnt/[beta]-catenin also showed abilities to inhibit the IL-17A-induced M1 macrophage polarization while diminishing the IL-17A-inhibited M2 polarization. Molecular analysis further uncovered that the JAK/STATsignaling pathway was involved in the interaction of Wnt/[beta]-catenin and IL-17A in the modulation of macrophage polarization. These results suggested that the Wnt/[beta]-catenin signaling modulated IL-17A-altered macrophage polarization in part by regulating the JAK/STAT signaling pathway. This study thus revealed a novel function of Wnt/[beta]-catenin signaling in regulating IL-17A-altered macrophage polarization. Keywords: Wnt/[beta]-catenin signaling; Interleukin-17A; Macrophage polarization; STAT; Immune regulation
Interleukins Analysis Macrophages

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