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Mutational Spectrum of DMD Mutations in Dystrophinopathy Patients: Application of Modern Diagnostic Techniques to a Large Cohort
Journal article   Open access   Peer reviewed

Mutational Spectrum of DMD Mutations in Dystrophinopathy Patients: Application of Modern Diagnostic Techniques to a Large Cohort

Kevin M Flanigan, Diane Dunn, Andrew von Niederhausern, Payam Soltanzadeh, Eduard Gappmaier, Michael T Howard, Jacinda Sampson, Jerry Mendell, Cheryl Wall, Wendy King, …
Human mutation, Vol.30(12), pp.1657-1666
12/2009
DOI: 10.1002/humu.21114
PMCID: PMC3404892
PMID: 19937601
url
https://doi.org/10.1002/humu.21114View
Published (Version of record) Open Access

Abstract

Mutations in the DMD gene, encoding the dystrophin protein, are responsible for the dystrophinopathies Duchenne Muscular Dystrophy (DMD), Becker Muscular Dystrophy (BMD), and X-linked Dilated Cardiomyopathy (XLDC). Mutation analysis has traditionally been challenging, due to the large gene size (79 exons over 2.2 Mb of genomic DNA). We report a very large aggregate data set comprised of DMD mutations detected in samples from patients enrolled in the United Dystrophinopathy Project, a multicenter research consortium, and in referral samples submitted for mutation analysis with a diagnosis of dystrophinopathy. We report 1111 mutations in the DMD gene, including 891 mutations with associated phenotypes. These results encompass 506 point mutations (including 294 nonsense mutations) and significantly expand the number of mutations associated with the dystrophinopathies, highlighting the utility of modern diagnostic techniques. Our data supports the uniform hypermutability of CGA>TGA mutations, establishes the frequency of polymorphic muscle (Dp427m) protein isoforms and reveals unique genomic haplotypes associated with `private' mutations. We note that 60% of these patients would be predicted to benefit from skipping of a single DMD exon using antisense oligonucleotide therapy, and 62% would be predicted to benefit from an inclusive multi-exon skipping approach directed toward exons 45 through 55.
BMD dystrophinopathy DMD Becker Muscular Dystrophy mutation detection Duchenne Muscular Dystrophy

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