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Nedd4 Controls Animal Growth by Regulating IGF-1 Signaling
Journal article   Peer reviewed

Nedd4 Controls Animal Growth by Regulating IGF-1 Signaling

Xiao R. Cao, Nancy L. Lill, Natasha Boase, Peijun P. Shi, David R. Croucher, Hongbo Shan, Jing Qu, Eileen M. Sweezer, Trenton Place, Patricia A. Kirby, …
Science signaling, Vol.1(38), pp.ra5-ra5
09/23/2008
DOI: 10.1126/scisignal.1160940
PMCID: PMC2833362
PMID: 18812566
url
http://doi.org/10.1126/scisignal.1160940View
Open Access

Abstract

The ubiquitin ligase Nedd4 has been proposed to regulate a number of signaling pathways, but its physiological role in mammals has not been characterized. Here we present an analysis of Nedd4-null mice to show that loss of Nedd4 results in reduced insulin-like growth factor 1 (IGF-1) and insulin signaling, delayed embryonic development, reduced growth and body weight, and neonatal lethality. In mouse embryonic fibroblasts, mitogenic activity was reduced, the abundance of the adaptor protein Grb10 was increased, and the IGF-1 receptor, which is normally present on the plasma membrane, was mislocalized. However, surface expression of IGF-1 receptor was restored in homozygous mutant mouse embryonic fibroblasts after knockdown of Grb10, and Nedd4(-/-) lethality was rescued by maternal inheritance of a disrupted Grb10 allele. Thus, in vivo, Nedd4 appears to positively control IGF-1 and insulin signaling partly through the regulation of Grb10 function.
Cell Biology Biochemistry & Molecular Biology Life Sciences & Biomedicine Science & Technology

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