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O-Mannosyl Phosphorylation of Alpha-Dystroglycan is Required for Laminin Binding
Journal article   Peer reviewed

O-Mannosyl Phosphorylation of Alpha-Dystroglycan is Required for Laminin Binding

Takako Yoshida-Moriguchi, Liping Yu, Stephanie H Stalnaker, Sarah Davis, Stefan Kunz, Michael B.A Oldstone, Harry Schachter, Lance Wells and Kevin P Campbell
Science (American Association for the Advancement of Science), Vol.327(5961), pp.88-92
01/01/2010
DOI: 10.1126/science.1180512
PMID: 20044576
url
https://iris.unil.ch/bitstreams/4f913c4c-0a58-4889-84e6-20c239ecc2dd/downloadView
Open Access

Abstract

Alpha-dystroglycan is a cell-surface glycoprotein that acts as a receptor for both extracellular matrix proteins containing laminin-G domains and certain arenaviruses. Receptor binding is thought to be mediated by a post-translational modification, and defective binding with laminin underlies a subclass of congenital muscular dystrophy. Here, using mass spectrometry- and NMR-based structural analyses, we identified a phosphorylated O -mannosyl glycan on the mucin-like domain of recombinant alpha-dystroglycan, which was required for laminin binding. We demonstrated that patients with muscle-eye-brain disease and Fukuyama congenital muscular dystrophy, as well as mice with myodystrophy, commonly have defects in a post-phosphoryl modification of this phosphorylated O -linked mannose, and that this modification is mediated by the like-acetylglucosaminyltransferase (LARGE) protein. Our findings expand our understanding of the mechanisms that underlie congenital muscular dystrophy.

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