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Patient-derived model capturing hypoxia and extracellular matrix remodelling of immunologically cold high-grade serous tumours
Journal article   Open access   Peer reviewed

Patient-derived model capturing hypoxia and extracellular matrix remodelling of immunologically cold high-grade serous tumours

Simona Plesselova, Hailey Axemaker, Kristin Calar, Oduduabasi Isaiah, Jared Wollman, Somshuvra Bhattacharya, Etienne Z Gnimpieba, Darci M Fink, Congzhou Wang, Hiruni Sumanasiri, …
Nature communications
07/11/2026
DOI: 10.1038/s41467-026-75262-8
PMID: 42436127
url
https://doi.org/10.1038/s41467-026-75262-8View
Published (Version of record) Open Access

Abstract

High-grade serous carcinoma tumours present poor survival rates, often associated with immunologically excluded environments driven by hypoxia and extensive extracellular matrix remodelling that disrupt tumour-stromal-immune interactions. Current experimental models fail to fully capture these microenvironmental features, limiting understanding of tumour-immune dynamics and drug development. Here, we present bioengineered patient-derived tumour-immune models to mimic physiologically relevant oxygen levels and extracellular matrix remodelling. Cancer cells are co-cultured with cancer-associated fibroblasts within human plasma-3D matrices or grown on decellularized human ovaries. Immune cells are either included within the 3D constructs to study multi-cellular interactions or challenged to infiltrate the matrices. We demonstrate that intratumoural hypoxia acts as a friend and a foe enhancing the activation and cytotoxicity of CD8 + T cells while inducing stromal/matrix dysregulation associated with impaired immune infiltration. Targeting TGF-β signalling attenuates the hypoxia-driven stromal-mediated immune exclusion. These relevant models may aid the development of targeted therapies to transform immunologically cold tumours into immunogenic to benefit female patients.

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