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Protective effects of dog allergen and endotoxin for lung function and asthma
Journal article   Peer reviewed

Protective effects of dog allergen and endotoxin for lung function and asthma

Makiko Nanishi, Myrtha E. Reyna, Marija Pajdakovska, Vanessa L. Breton, Marc-Antoine Bédard, Qingling Duan, Elizabeth George MBI, Mohammad Kaviul Anam Khan, Biswajit Chowdhury, Bree T. Sharma, …
Journal of allergy and clinical immunology
02/23/2026
DOI: 10.1016/j.jaci.2026.02.016
PMID: 41740928

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Abstract

Background Childhood asthma is characterized by altered lung function and airway inflammation, thought to result from complex gene-environment interactions, especially with allergens. However, previous studies show inconsistent associations between allergen exposure and asthma. Objectives We aimed to examine the longitudinal relationship between indoor allergen exposure during infancy with subsequent asthma and spirometry and the potential effect modification by genetic factors. Methods Data from a subcohort of the CHILD study with analyzed dust samples (including Can f1 (dog), Fel d1 (cat), and endotoxin) and physician diagnosed asthma or spirometry were used to examine the relationships between allergen levels in dust analytes at 3 months and asthma and, separately, spirometry at 5 years, including potential effect modification by genetic factors using lung function polygenic scores (PGSs). Results Among 1050 children with dust samples, 6.6% developed asthma by age 5 years. In an adjusted multivariable model, higher Can f1 significantly decreased the risk of asthma (OR 0.52, 95% CI 0.25, 0.98). Independently, children exposed to high Can f1 had significantly higher FEV1 z-scores (β=0.23, 95% CI 0.06, 0.40), regardless of asthma status. In the gene environment analyses, there were significant gene-environment interactions in the relationship between Can f1 and PGSs on lung function, independent of asthma status. Conclusions In a general population birth cohort, early-life exposure to high levels of Can f1 was associated with improved lung function and protection against asthma at age 5. Furthermore, exposure to high Can f1 may modulate lung function in individuals with low PGSs.
Asthma children early-life indoor allergens endotoxin gene-environment interactions lung function

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