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Protein kinase D regulates basolateral membrane protein exit from trans -Golgi network
Journal article   Peer reviewed

Protein kinase D regulates basolateral membrane protein exit from trans -Golgi network

Vivek Malhotra, M. Inmaculada Ayala, Carine Bossard, Charles Yeaman, Agnes Ang, Jessica R Wright, W. James Nelson, Thomas Seufferlein, Ira Mellman, Frederic Bard, …
Nature cell biology, Vol.6(2), pp.106-112
02/2004
DOI: 10.1038/ncb1090
PMCID: PMC3372901
PMID: 14743217

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Abstract

Protein kinase D (PKD) binds to diacylglycerol (DAG) in the trans-Golgi network (TGN) and is activated by trimeric G-protein subunits βγ. This complex then regulates the formation of transport carriers in the TGN that traffic to the plasma membrane in non-polarized cells. Here we report specificity of different PKD isoforms in regulating protein trafficking from the TGN. Kinase-inactive forms of PKD1, PKD2 and PKD3 localize to the TGN in polarized and non-polarized cells. PKD activity is required only for the transport of proteins containing basolateral sorting information, and seems to be cargo specific.

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