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Protons are a neurotransmitter that regulates synaptic plasticity in the lateral amygdala
Journal article   Open access   Peer reviewed

Protons are a neurotransmitter that regulates synaptic plasticity in the lateral amygdala

Jianyang Du, Leah R Reznikov, Margaret P Price, Xiang-ming Zha, Yuan Lu, Thomas O Moninger, John A Wemmie and Michael J Welsh
Proceedings of the National Academy of Sciences - PNAS, Vol.111(24), pp.8961-8966
06/17/2014
DOI: 10.1073/pnas.1407018111
PMCID: PMC4066526
PMID: 24889629
url
https://doi.org/10.1073/pnas.1407018111View
Published (Version of record) Open Access

Abstract

Synaptic transmission involves the release of neurotransmitters that activate receptors on postsynaptic cells. The results reveal that protons fulfill the criteria for a neurotransmitter and that they activate postsynaptic acid-sensing ion channels. This activity facilitates synaptic plasticity, a requirement for learning and memory in the amygdala. Stimulating presynaptic terminals can increase the proton concentration in synapses. Potential receptors for protons are acid-sensing ion channels (ASICs), Na + - and Ca 2+ -permeable channels that are activated by extracellular acidosis. Those observations suggest that protons might be a neurotransmitter. We found that presynaptic stimulation transiently reduced extracellular pH in the amygdala. The protons activated ASICs in lateral amygdala pyramidal neurons, generating excitatory postsynaptic currents. Moreover, both protons and ASICs were required for synaptic plasticity in lateral amygdala neurons. The results identify protons as a neurotransmitter, and they establish ASICs as the postsynaptic receptor. They also indicate that protons and ASICs are a neurotransmitter/receptor pair critical for amygdala-dependent learning and memory.
acid sensing ion channel Biological Sciences long-term potentiation PcTX1

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