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Restoring Acid-Sensing Ion Channel-1a in the Amygdala of Knock-Out Mice Rescues Fear Memory But Not Unconditioned Fear Responses
Journal article   Open access   Peer reviewed

Restoring Acid-Sensing Ion Channel-1a in the Amygdala of Knock-Out Mice Rescues Fear Memory But Not Unconditioned Fear Responses

Matthew W Coryell, Amanda M Wunsch, Jill M Haenfler, Jason E Allen, Jodi L McBride, Beverly L Davidson and John A Wemmie
The Journal of neuroscience, Vol.28(51), pp.13738-13741
12/17/2008
DOI: 10.1523/JNEUROSCI.3907-08.2008
PMID: 19091964
url
https://doi.org/10.1523/JNEUROSCI.3907-08.2008View
Published (Version of record) Open Access

Abstract

Acid-sensing ion channel-1a (ASIC1a) contributes to multiple fear behaviors, however the site of ASIC1a action in behavior is not known. To explore a specific location of ASIC1a action, we expressed ASIC1a in the basolateral amygdala of ASIC1a–/– mice using viral vector-mediated gene transfer. This rescued context-dependent fear memory, but not the freezing deficit during training or the unconditioned fear response to predator odor. These data pinpoint the basolateral amygdala as the site where ASIC1a contributes to fear memory. They also discriminate fear memory from fear expressed during training and from unconditioned fear. Furthermore, this work illustrates a strategy for identifying discrete brain regions where specific genes contribute to complex behaviors.
ASIC1a region-restricted amygdala fear conditioning Brief Communications unconditioned fear viral-vector-mediated gene transfer

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