Journal article
Rubral lateropulsion due to vertebral artery dissection in a patient with Klippel-Feil syndrome
Archives of neurology (Chicago), Vol.61(4), pp.583-585
2004
DOI: 10.1001/archneur.61.4.583
PMID: 15096409
Abstract
Background: Neurologic deficits in patients with Klippel-Feil syndrome usually are attributed to direct compression of neuronal structures or hypoperfusion secondary to compression of the vertebral arteries by bony abnormalities.
Objective: To describe a 38-year-old woman with known Klippel-Feil syndrome who developed lateropulsion.
Results: The results of magnetic resonance imaging were consistent with rubrothalamic stroke. The cerebral angiogram confirmed vertebral artery dissection at the level of her previously observed bony abnormality.
Conclusions: Hypermobility adjacent to fused vertebrae subjects the vertebral artery to increased shear forces. Thus, Klippel-Feil syndrome may be a predisposing factor for vertebral artery dissection. Moreover, to our knowledge, this case represents the second known case of rubral lateropulsion.
Details
- Title: Subtitle
- Rubral lateropulsion due to vertebral artery dissection in a patient with Klippel-Feil syndrome
- Creators
- Morvarid KARIMI - Department of Neurology, University of Iowa Hospitals and Clinics, Iowa City, United StatesMehrdad RAZAVI - Department of Neurology, University of Iowa Hospitals and Clinics, Iowa City, United StatesDeema FATTAL - Department of Neurology, University of Iowa Hospitals and Clinics, Iowa City, United States
- Resource Type
- Journal article
- Publication Details
- Archives of neurology (Chicago), Vol.61(4), pp.583-585
- DOI
- 10.1001/archneur.61.4.583
- PMID
- 15096409
- NLM abbreviation
- Arch Neurol
- ISSN
- 0003-9942
- eISSN
- 1538-3687
- Publisher
- American Medical Association; Chicago, IL
- Language
- English
- Date published
- 2004
- Academic Unit
- Neurology
- Record Identifier
- 9984020869102771
Metrics
19 Record Views