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TOR Signaling Pathway in Cardiac Aging and Heart Failure
Journal article   Open access   Peer reviewed

TOR Signaling Pathway in Cardiac Aging and Heart Failure

Nastaran Daneshgar, Peter S Rabinovitch and Dao-Fu Dai
Biomolecules (Basel, Switzerland), Vol.11(2), p.168
01/27/2021
DOI: 10.3390/biom11020168
PMID: 33513917
url
https://doi.org/10.3390/biom11020168View
Published (Version of record) Open Access

Abstract

Mechanistic Target of Rapamycin (mTOR) signaling is a key regulator of cellular metabolism, integrating nutrient sensing with cell growth. Over the past two decades, studies on the mTOR pathway have revealed that mTOR complex 1 controls life span, health span, and aging by modulating key cellular processes such as protein synthesis, autophagy, and mitochondrial function, mainly through its downstream substrates. Thus, the mTOR pathway regulates both physiological and pathological processes in the heart from embryonic cardiovascular development to maintenance of cardiac homeostasis in postnatal life. In this regard, the dysregulation of mTOR signaling has been linked to many age-related pathologies, including heart failure and age-related cardiac dysfunction. In this review, we highlight recent advances of the impact of mTOR complex 1 pathway and its regulators on aging and, more specifically, cardiac aging and heart failure.
heart failure caloric restriction cardiac aging mTOR Review aging rapamycin

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