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Targeting Endoplasmic Reticulum Stress as an Effective Treatment for Alcoholic Pancreatitis
Journal article   Open access   Peer reviewed

Targeting Endoplasmic Reticulum Stress as an Effective Treatment for Alcoholic Pancreatitis

Hui Li, Wen Wen and Jia Luo
Biomedicines, Vol.10(1), p.108
01/05/2022
DOI: 10.3390/biomedicines10010108
PMCID: PMC8773075
PMID: 35052788
url
https://doi.org/10.3390/biomedicines10010108View
Published (Version of record) Open Access

Abstract

Pancreatitis and alcoholic pancreatitis are serious health concerns with an urgent need for effective treatment strategies. Alcohol is a known etiological factor for pancreatitis, including acute pancreatitis (AP) and chronic pancreatitis (CP). Excessive alcohol consumption induces many pathological stress responses; of particular note is endoplasmic reticulum (ER) stress and adaptive unfolded protein response (UPR). ER stress results from the accumulation of unfolded/misfolded protein in the ER and is implicated in the pathogenesis of alcoholic pancreatitis. Here, we summarize the possible mechanisms by which ER stress contributes to alcoholic pancreatitis. We also discuss potential approaches targeting ER stress and UPR in developing therapeutic strategies for the disease.
FDA-approved drugs alcohol abuse therapy oxidative stress cell signaling

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