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Temporally Evolving and Context-Dependent Functions of Cytokines That Regulate Murine Anti- Plasmodium Humoral Immunity
Journal article   Open access   Peer reviewed

Temporally Evolving and Context-Dependent Functions of Cytokines That Regulate Murine Anti- Plasmodium Humoral Immunity

Fionna A Surette and Noah S Butler
Pathogens (Basel), Vol.11(5), p.523
04/29/2022
DOI: 10.3390/pathogens11050523
PMCID: PMC9144513
PMID: 35631044
url
https://doi.org/10.3390/pathogens11050523View
Published (Version of record) Open Access

Abstract

Protective immunity against blood-stage infection and the disease malaria depends on antibodies secreted from high-affinity B cells selected during the germinal center (GC) response. The induction and stability of the GC response require the activation and direct cell-cell communication between parasite-specific CD4 helper T cells and B cells. However, cytokines secreted by helper T cells, B cells, and multiple other innate and adaptive immune cells also contribute to regulating the magnitude and protective functions of GC-dependent humoral immune responses. Here, we briefly review emerging data supporting the finding that specific cytokines can exhibit temporally distinct and context-dependent influences on the induction and maintenance of antimalarial humoral immunity.
type I interferon B cell Plasmodium CD4 T cell germinal center T follicular helper malaria interleukin-10 interleukin-21

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