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The PDGF-C regulatory region SNP rs28999109 decreases promoter transcriptional activity and is associated with CL/P
Journal article   Open access   Peer reviewed

The PDGF-C regulatory region SNP rs28999109 decreases promoter transcriptional activity and is associated with CL/P

Sun J CHOI, Mary L MARAZITA, Somnya NARAYANAN, Maria Adela Mansilla, José M GRANJEIRO, Alexandre R VIEIRA, Andrew C LIDRAL, Jeffrey C MURRAY, Thomas C HART, P SUZANNE HART, …
European journal of human genetics : EJHG, Vol.17(6), pp.774-784
2009
DOI: 10.1038/ejhg.2008.245
PMCID: PMC2788748
PMID: 19092777
url
https://doi.org/10.1038/ejhg.2008.245View
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Abstract

Human linkage and association studies suggest a gene(s) for nonsyndromic cleft lip with or without cleft palate (CL/P) on chromosome 4q31–q32 at or near the platelet-derived growth factor-C (PDGF-C) locus. The mouse Pdgfc−/− knockout shows that PDGF-C is essential for palatogenesis. To evaluate the role of PDGF-C in human clefting, we performed sequence analysis and SNP genotyping using 1048 multiplex CL/P families and 1000 case–control samples from multiple geographic origins. No coding region mutations were identified, but a novel −986 C>T SNP (rs28999109) was significantly associated with CL/P (P=0.01) in cases from Chinese families yielding evidence of linkage to 4q31–q32. Significant or near-significant association was also seen for this and several other PDGF-C SNPs in families from the United States, Spain, India, Turkey, China, and Colombia, whereas no association was seen in families from the Philippines, and Guatemala, and case–controls from Brazil. The −986T allele abolished six overlapping potential transcription regulatory motifs. Transfection assays of PDGF-C promoter reporter constructs show that the −986T allele is associated with a significant decrease (up to 80%) of PDGF-C gene promoter activity. This functional polymorphism acting on a susceptible genetic background may represent a component of human CL/P etiology.
Medical Genetics Fundamental and applied biological sciences. Psychology General aspects. Genetic counseling Biological and medical sciences Molecular and cellular biology Genetics of eukaryotes. Biological and molecular evolution Medical sciences

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