Journal article
The changing concept of cardiovascular reactivity
Stress medicine, Vol.4(4), pp.241-251
10/1988
DOI: 10.1002/smi.2460040409
Abstract
Cardiovascular reactivity has long been measured in laboratory responses to a wide variety of cognitive and physical stressors, with the hypothesis that exaggerated reactivity plays a pathogenetic role in the development of essential hypertension. However there are few data to support the belief that behavioural differences of cognitive perception of stressors account for observed differences of reactivity. Cardiovascular reactivity in the laboratory does not predict hypertension or account for differences of blood pressure variability in the natural environment. Hypertensives do not exhibit increased blood pressure variability. Antihypertensive therapy consistently fails to lower cardiovascular reactivity in either the laboratory or natural milieu, supporting the dual and largely independent regulation of the basal and reactive blood pressures. It is concluded that there is little support for the use of laboratory stress testing to delineate either the pathogenesis of hypertension, the evaluation of hypertensive subjects, or the efficacy of antihypertensive therapy. Copyright © 1988 John Wiley & Sons, Ltd
Details
- Title: Subtitle
- The changing concept of cardiovascular reactivity
- Creators
- Ray H Rosenman - Health Sciences Program, SRI International, 323 Ravenswood Avenue, Menlo Park, CA 94025, USAMarcia M Ward - Health Sciences Program, SRI International, 323 Ravenswood Avenue, Menlo Park, CA 94025, USA
- Resource Type
- Journal article
- Publication Details
- Stress medicine, Vol.4(4), pp.241-251
- Publisher
- John Wiley & Sons, Ltd
- DOI
- 10.1002/smi.2460040409
- ISSN
- 0748-8386
- eISSN
- 1099-1700
- Number of pages
- 11
- Language
- English
- Date published
- 10/1988
- Academic Unit
- Health Management and Policy
- Record Identifier
- 9984214672502771
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