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The molecular basis of selective DNA binding by the BRG1 AT-hook and bromodomain
Journal article   Open access   Peer reviewed

The molecular basis of selective DNA binding by the BRG1 AT-hook and bromodomain

Julio C Sanchez, Liyang Zhang, Stefania Evoli, Nicholas J Schnicker, Maria Nunez-Hernandez, Liping Yu, Jeff Wereszczynski, Miles A Pufall and Catherine A Musselman
Biochimica et biophysica acta. Gene regulatory mechanisms, Vol.1863(8), pp.194566-194566
08/2020
DOI: 10.1016/j.bbagrm.2020.194566
PMID: 32376391
url
https://doi.org/10.1016/j.bbagrm.2020.194566View
Published (Version of record) Open Access

Abstract

The ATP-dependent BAF chromatin remodeling complex plays a critical role in gene regulation by modulating chromatin architecture, and is frequently mutated in cancer. Indeed, subunits of the BAF complex are found to be mutated in >20% of human tumors. The mechanism by which BAF properly navigates chromatin is not fully understood, but is thought to involve a multivalent network of histone and DNA contacts. We previously identified a composite domain in the BRG1 ATPase subunit that is capable of associating with both histones and DNA in a multivalent manner. Mapping the DNA binding pocket revealed that it contains several cancer mutations. Here, we utilize SELEX-seq to investigate the DNA specificity of this composite domain and NMR spectroscopy and molecular modelling to determine the structural basis of DNA binding. Finally, we demonstrate that cancer mutations in this domain alter the mode of DNA association. •The BRG1 AT-BD binds multivalently to DNA.•The BRG1 AT-BD is selective for AATTAAAT containing sequences.•The BRG1 AT-BD spans the minor and major grooves of DNA.

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