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The pathophysiology of acute pain: animal models
Journal article   Open access

The pathophysiology of acute pain: animal models

Jun Xu and Timothy J. Brennan
Current opinion in anaesthesiology, Vol.24(5), pp.508-514
10/01/2011
DOI: 10.1097/ACO.0b013e32834a50d8
PMCID: PMC3259564
PMID: 21841478
url
https://www.ncbi.nlm.nih.gov/pmc/articles/3259564View
Open Access

Abstract

Purpose of review Trauma, surgery, and burns are three common clinical scenarios that are associated with significant acute pain. This review describes the pathophysiology of acute pain utilizing three preclinical models: surgery, burn, and fracture. Recent findings In general, there is greater interest directed toward peripheral mediators of acute pain. Studies indicate that treatment against nerve growth factor, interleukins, and ischemic-like mediators may provide valuable avenues for treatment of acute pain. By targeting the periphery, analgesic therapies may have reduced side-effects. Summary Peripheral mediators of acute pain can vary depending upon the type of injury. Treatment aimed toward those mediators specific to the injury may improve acute pain management in the future. It will be important to translate these findings into clinical trials in the future.
Anesthesiology Life Sciences & Biomedicine Science & Technology

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