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Three months of high-fructose feeding fails to induce excessive weight gain or leptin resistance in mice
Journal article   Open access   Peer reviewed

Three months of high-fructose feeding fails to induce excessive weight gain or leptin resistance in mice

Erik J Tillman, Donald A Morgan, Kamal Rahmouni and Steven J Swoap
PloS one, Vol.9(9), pp.e107206-e107206
2014
DOI: 10.1371/journal.pone.0107206
PMCID: PMC4161399
PMID: 25211467
url
https://doi.org/10.1371/journal.pone.0107206View
Published (Version of record) Open Access

Abstract

High-fructose diets have been implicated in obesity via impairment of leptin signaling in humans and rodents. We investigated whether fructose-induced leptin resistance in mice could be used to study the metabolic consequences of fructose consumption in humans, particularly in children and adolescents. Male C57Bl/6 mice were weaned to a randomly assigned diet: high fructose, high sucrose, high fat, or control (sugar-free, low-fat). Mice were maintained on their diets for at least 14 weeks. While fructose-fed mice regularly consumed more kcal and expended more energy, there was no difference in body weight compared to control by the end of the study. Additionally, after 14 weeks, both fructose-fed and control mice displayed similar leptin sensitivity. Fructose-feeding also did not change circulating glucose, triglycerides, or free fatty acids. Though fructose has been linked to obesity in several animal models, our data fail to support a role for fructose intake through food lasting 3 months in altering of body weight and leptin signaling in mice. The lack of impact of fructose in the food of growing mice on either body weight or leptin sensitivity over this time frame was surprising, and important information for researchers interested in fructose and body weight regulation.
High Fructose Corn Syrup - administration & dosage Administration, Oral Mice, Inbred C57BL Diet, High-Fat - adverse effects Male Obesity - metabolism Leptin - physiology Animals Diet Basal Metabolism Female Obesity - etiology Weight Gain

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